Compound · kpv
T3Peptide

KPV

Tripeptide fragment (Lys-Pro-Val) derived from the C-terminal end of alpha-melanocyte-stimulating hormone (alpha-MSH). Retains the anti-inflammatory properties of alpha-MSH without melanocortin receptor activation. Inhibits NF-kB pathway and reduces pro-inflammatory cytokine production. Crosses mucosal barriers with demonstrated efficacy in colitis models.

Half-life
Short (tripeptide, rapid degradation)
Bioavailability
Oral (mucosal absorption); subcutaneous (systemic)
Route
oral, subcutaneous
Evidence tier
T3 — Community / emerging
Optimization pillars
recovery
References
2 peer-reviewed
Dose ranges

Three tiers ordered by aggressiveness. Tier chips on every OPTIMIZE intervention let you filter the catalog by your evidence tolerance.

conservative
200–500 mcg/day
Mild anti-inflammatory support
moderate
500–1000 mcg/day
Gut inflammation
aggressive
1000–1500 mcg/day
Active inflammatory condition
Monitoring
  • hs-crp
  • wbc
Contraindications
  • pregnancy
References
  • PMID:15249167Systemic administration of alpha-MSH analogs inhibits gastrointestinal inflammationNeuropeptides, 2004
  • PMID:25398442Anti-inflammatory effects of alpha-MSH peptide fragments in intestinal epithelial cellsPeptides, 2015
Notes

KPV is the stripped-down anti-inflammatory fragment. Three amino acids from alpha-MSH that retain the NF-kB suppression without the melanocortin receptor effects. No tanning. No sexual side effects. Just the inflammatory shutdown. The oral route for gut-specific inflammation is the most logical application — a tripeptide that crosses the mucosal barrier to suppress inflammation locally. The colitis model data is encouraging. The human clinical data is absent. This is a T3 compound that may eventually move to T2 as the IBD research catches up.

This is not medical advice

Discuss with a licensed clinician before starting, stopping, or changing any compound. This page documents what the research literature describes — it is not a prescription.

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