Compound · ll-37
T3Peptide

LL-37

37-amino acid human cathelicidin antimicrobial peptide. The only cathelicidin found in humans. Disrupts bacterial membranes via electrostatic interaction with lipid bilayers. Also modulates innate immune response — recruits immune cells, promotes angiogenesis, and has anti-biofilm activity. Active against bacteria, fungi, and some viruses.

Half-life
Poorly characterized systemically (local tissue persistence)
Bioavailability
Local via subcutaneous; systemic distribution limited
Route
subcutaneous
Evidence tier
T3 — Community / emerging
Optimization pillars
recovery
References
2 peer-reviewed
Dose ranges

Three tiers ordered by aggressiveness. Tier chips on every OPTIMIZE intervention let you filter the catalog by your evidence tolerance.

conservative
50–100 mcg/day
Immune modulation
moderate
100–200 mcg/day
Antimicrobial support
aggressive
200–400 mcg/day
Active infection adjunct
Monitoring
  • wbc
  • hs-crp
Contraindications
  • pregnancy
  • autoimmune-disease
References
  • PMID:16278835Antimicrobial peptides and the innate immune responsePeptides, 2006
  • PMID:22110885LL-37, the only human member of the cathelicidin family of antimicrobial peptidesBiochim Biophys Acta, 2012
Notes

LL-37 is your body's endogenous antibiotic. It punches holes in bacterial membranes. It disrupts biofilms — the slime-fortress structures that make chronic infections resistant to conventional antibiotics. The anti-biofilm property is the most interesting clinical angle. Chronic sinusitis, SIBO, persistent Lyme — conditions where biofilm formation is the therapeutic bottleneck. The evidence for exogenous LL-37 injection in these contexts is preclinical. The mechanism is sound. The clinical data is not there yet.

This is not medical advice

Discuss with a licensed clinician before starting, stopping, or changing any compound. This page documents what the research literature describes — it is not a prescription.

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