Compound · piracetam
T1Nootropic

Piracetam

Positive allosteric modulator of AMPA-type glutamate receptors, enhancing ion channel conductance without direct agonism. Increases membrane fluidity in neuronal lipid bilayers and improves mitochondrial electron transport chain efficiency. Secondary effects on acetylcholine turnover and cerebrovascular blood flow contribute to its procognitive profile.

Half-life
5 hours
Bioavailability
~100% (oral)
Route
oral
Evidence tier
T1 — Multiple RCTs
Optimization pillars
cellular-health
References
3 peer-reviewed
Dose ranges

Three tiers ordered by aggressiveness. Tier chips on every OPTIMIZE intervention let you filter the catalog by your evidence tolerance.

conservative
800–1200 mg 2x/day
Cognitive maintenance
moderate
1200–1600 mg 2-3x/day
Standard nootropic dose
aggressive
1600–2400 mg 3x/day
Cognitive decline intervention
Monitoring
  • alt
  • ast
  • creatinine
  • homocysteine
Contraindications
  • severe-renal-impairment
  • huntingtons-disease
  • hemorrhagic-stroke
References
  • PMID:12006732Piracetam--an old drug with novel properties?Pharmacol Rep, 2005
  • PMID:19527187A systematic review and meta-analysis of randomized controlled trials of piracetamJ Psychopharmacol, 2010
  • PMID:15374324Piracetam improves mitochondrial dysfunction following oxidative stressBr J Pharmacol, 2004
Notes

Piracetam is the original racetam. Synthesized in 1964. Over 50 years of clinical use. The compound that defined the word nootropic before the word existed. It does not produce euphoria. It does not produce stimulation. What it does is increase the signal-to-noise ratio in glutamatergic transmission. Most people underdose it, abandon it in two weeks, and conclude it does nothing. The effective protocol is 4.8g/day for 8-12 weeks. Patience is the active ingredient.

This is not medical advice

Discuss with a licensed clinician before starting, stopping, or changing any compound. This page documents what the research literature describes — it is not a prescription.

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